King's College London Network Centre
The Network is designed to support dementia research through the funding of collaborative science and networking to share findings and resources.
Here are just a handful of recent publications from within the King’s College London Research Network Centre.
- Direct Keap1-Nrf2 disruption as a potential therapeutic target for Alzheimer’s disease, Kerr et al. (2017)
- Membrane association and release of wild-type and pathological tau from organotypic brain slice cultures, CL Croft et al. (2017)
- Roles of tau protein in health and disease, T Guo et al. (2017)
- α-Synuclein binds to the ER-mitochondria tethering protein VAPB to disrupt Ca2+ homeostasis and mitochondrial ATP production, S Paillusson et al. (2017)
- The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy, P Gomez-Suaga et al. (2017)
- Mutations in ANXA11 cause familial and sporadic amyotrophic lateral sclerosis, BN Smith et al. (2017)
- Clusterin expression is upregulated following acute head injury and localizes to astrocytes in old head injury, C Troakes et al. (2017)
- Aluminium in brain tissue in familial Alzheimer’s disease, A Mirza et al. (2017)
- A dysfunctional blood-brain barrier and cerebral small vessel disease, AH Hainsworth et al. (2017)
- Translational models for vascular cognitive impairment: a review including larger species, AH Hainsworth et al. (2017)
- Bencze et al. (2018) Biological function of Lemur tyrosine kinase 2 (LMTK2): implications in neurodegeneration. Mol. Brain 11, 20, doi:10.1186/s13041-018-0363-x.
- Gomez-Suaga et al. (2019) The VAPB-PTPIP51 endoplasmic reticulum-mitochondria tethering proteins are present in neuronal synapses and regulate synaptic activity. Acta Neuropath.Commun. 7, 35, doi:10.1186/s40478-019-0688-4.
- Ashraf et al (2019). Pattern of altered plasma elemental phosphorous, calcium, selenium, iron and copper in Alzheimer’s Disease. Sci. Rep. 9: 3147.
- Mórotz et al. (2019) LMTK2 binds to kinesin light chains to mediate anterograde axonal transport of cdk5/p35 andLMTK2 levels are reduced in Alzheimer’s disease brains. Acta Neuropath. Commun. 7, 73, doi:10.1186/s40478-019-0715-5.
If you have equipment and resources you’d like shared with the Network Centre, get in touch.